Kenneth  C.  Bilchick
Degree(s): MD, MS
Graduate School: Johns Hopkins University School of Medicine
Primary Appointment: Assistant Professor, Medicine, Cardiovascular Medicine
Research Interests:
Cardiac resynchronization, cardiac MRI, heart failure, atrial fibrillation, ventricular tachycardia, ICDs
Email Address: kcb7f@virginia.edu

Research Description

Dr. Bilchick’s research includes:

· the role of cardiac magnetic resonance therapy in patients with heart failure referred for cardiac resynchronization therapy.
· the application of cardiac magnetic resonance imaging to patients undergoing cardiac resynchronization therapy.
· regional stress-induced gene expression changes in cardiac dyssynchrony, hemodynamic effects of HF-associated alterations in troponin I phosphorylation, pulmonary vein isolation for atrial fibrillation, and assessment of regional strain and function in heart failure with cardiac magnetic resonance imaging (CMR) myocardial tissue tagging and/or strain encoding (SENC).


Selected Publications
  • Bilchick KC, Lardo AC. Cardiac resynchronization therapy: application of imaging to optimize patient selection and assess response. Current Heart Failure Reports 2008; 5(3):119-27.
  • Bilchick KC, Dimaano V, Wu KC, Helm RH, Weiss RG, Lima JA, Berger RD, Tomaselli GT, Bluemke DA, Halperin HR, Abraham T, Kass DA, Lardo AC. Magnetic resonance imaging analysis of dyssynchrony and myocardial scar predicts function class improvement following cardiac resynchronization therapy. JACC Cardiovascular Imaging 2008; 1(5): 561-8.
  • Bilchick KC, Duncan JG, Ravi R, Takimoto E, Champion HC, Gao WD, Stull LB, Kass DA, Murphy AM. Heart failure-associated alterations in troponin I phosphorylation impair ventricular relaxation after load and force-frequency responses and systolic function. American Journal of Physiology: Heart and Circulatory Physiology 2007; 292(1):H318-25.
  • Bilchick KC, Saha SK, Mikolajczyk E, Cope L, Ferguson WJ, Yu W, Girouard S, Kass DA. Differential regional gene expression from cardiac dyssynchrony induced by chronic right ventricular free wallpacing in the mouse. Physiological Genomics 2006; 26(2):109-15.
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